Semax is a synthetic peptide derived from a fragment of adrenocorticotropic hormone (ACTH 4–10), modified to enhance stability and duration of activity. In research environments, Semax is studied for its involvement in neurotrophic signaling, synaptic plasticity pathways, and cognitive-related processes. It is commonly explored in neuroscience research due to its influence on brain-derived signaling mechanisms without exhibiting hormonal activity associated with full-length ACTH.
